By Shanda H. Blackmon, Min P. Kim, Karen J. Dickinson

This atlas presents a complete, cutting-edge evaluation of all interventions that pertain to the esophagus. It encompasses a overview of the present staging modalities, ablation applied sciences, resection and reconstruction ideas, and sickness class. Evidence-based instructions concerning how each one intervention is selected also are integrated. With colour illustrations and images for every surgical procedure, the atlas information particular anatomic issues reminiscent of micro-anatomy of Barrett’s and Dysplasia, EMR pathology, endoscopic ultrasound, and standard surgical anatomy. every one intervention is gifted in activity layout as a role record to be checked-off as every one step is completed.

Written by way of specialists within the box, Atlas of Esophageal disorder and Intervention: A Multidisciplinary Approach serves as a worthwhile source for any practitioner who plays esophageal intervention and should consultant new surgeons and gastroenterologists into the hybrid multidisciplinary method of this disease.

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Extra info for Atlas of Esophageal Disease and Intervention: A Multidisciplinary Approach

Sample text

24). This finding may mimic the perinuclear halo seen with the human papillomavirus cytopathic effect. The absence of characteristic nuclear changes supports a diagnosis of glycogen accumulation. a b Fig. 24 (a) Glycogen accumulation (H & E, ×100). (b) Glycogen accumulation (PAS without diastase, ×100). The PAS stain highlights the cytoplasmic glycogen 2 Histologic Anatomy 49 Herpes Esophagitis Herpesvirus infects the squamous epithelium of the esophagus, leading to ulceration (Fig. 25). The ulcer has a purulent base and well-demarcated edges.

The squamous epithelial cells show reactive features including nuclear enlargement and hyperchromasia.

B) Candida esophagitis (H & E, ×400) 42 B. Gorman Eosinophilic Esophagitis In eosinophilic esophagitis, a myriad of eosinophils infiltrates the epithelium and the lamina propria (Fig. 18). Intraepithelial clusters of eosinophils (eosinophilic abscesses) and degranulation are characteristic. Features of reflux esophagitis are also seen (basal cell hyperplasia, dilated intercellular spaces, and elongation of the lamina propria papillae) [8]. Scattered eosinophils may be present in reflux esophagitis, but the amount seen in eosinophilic esophagitis is far greater.

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